000			01997nam a22002537a 4500
999			_c8085 _d8085
001			CH 2009 0005
003			PILC
005			20240720152613.0
008			160505b2009 xxu||||| |||| 00| 0 eng d
040			_beng _cFEU-NRMF MEDICAL LIBRARY _erda
041			_aenglish
050			_aCH 2009 0005
245			_aDrug induced hepatitis in a patient with disseminated tuberculosis : _ba case report / _cLea G. Galia.
260			_aFairview, Quezon City: _bDepartment of Child Health, FEU-NRMF, _c2009.
300			_btables; _c(in folder)
336			_atext _2rdacontent
337			_aunmediated _2rdamedia
338			_avolume _2rdacarrier
504			_aIncludes appendices and bibliographical references.
520			_aAbstract: Presented was a case of a male adolescent with pulmonary tuberculosis undergoing treatment for 5 weeks with antituberculosis medications who suddenly developed jaundice and icteric sclerae. During the course of illness, patient presented with changes in sensorium and had highly elevated level of plasma ammonia causing encephalopathy. What could have caused the sudden onset of the above signs and symptoms? Could this be a case of viral hepatitis? Or a case of a drug induced hepatitis? Or a case of tuberculous meningitis? Further workup revealed the presence of granulomatous lesions to extrapulmonary sites specifically the liver and spleen thus gaining a presumptive diagnosis of disseminated tuberculosis. Tuberculosis (TB) is a serious disease of global importance, with a rising incidence in the developed world in recent years. The diagnosis of non-pulmonary TB poses a particular challenge for clinicians because of the protean ways in which the disease presents. Lymphohematogeneous spread always presents as the major pathway of spread of extra pulmonary tuberculosis. The usual progression is for the disease to spread from the lungs to the extrapulmonary sites.
521			_aRESDCH
700			_aGalia, Lea G., MD. _eauthor
942			_2lcc _cRU