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A 57-year-old male with Pharyngeal-Cervical-Brachial variant of Guillain-Barre Syndrome: A case report / [author]: Natasha Elaine L. Pecache,

Contributor(s): Publication details: Fairview, Quezon city; Department of Internal Medicine, FEU-NRMF, 2024Description: ( in folder) with flash drive (soft copy)Content type:
  • text
Media type:
  • unmediated
Carrier type:
  • volume
LOC classification:
  • MED 2024 0010
Online resources: Summary: ABSTRACT Guillain-Barre syndrome (GBS) is an acute, rare, frequently severe, and fulminant post-infectious polyradiculoneuropathy that is autoimmune in nature. It results from the autoimmune destruction of nerves in the peripheral nervous system causing symptoms such as numbness, tingling, and weakness that can progress to paralysis. In this report, we describe an adult man who consulted due to severe headache, odynophagia, dysphagia, dysarthria, loss of balance, ophthalmalgia, blurring of vision, and bilateral ptosis. During the course of his admission, he had symptoms of persistent headache, dysphagia, dysarthria, complete 3rd nerve palsy, and upper limb weakness. Further workup confirmed that he has a sensorimotor, primarily demyelinating, polyneuropathy leading to a diagnosis of Guillain Barre’ Syndrome should be a standard in patients who present with polyneuropathy. Early recognition and diagnosis will guide us to the proper management and help improve the patient and their family’s quality of time.
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Research Far Eastern University - Nicanor Reyes Medical Foundation MED 2024 0010 (Browse shelf(Opens below)) Available R000791

Includes bibliographical references

ABSTRACT
Guillain-Barre syndrome (GBS) is an acute, rare, frequently severe, and fulminant post-infectious polyradiculoneuropathy that is autoimmune in nature. It results from the autoimmune destruction of nerves in the peripheral nervous system causing symptoms such as numbness, tingling, and weakness that can progress to paralysis. In this report, we describe an adult man who consulted due to severe headache, odynophagia, dysphagia, dysarthria, loss of balance, ophthalmalgia, blurring of vision, and bilateral ptosis. During the course of his admission, he had symptoms of persistent headache, dysphagia, dysarthria, complete 3rd nerve palsy, and upper limb weakness. Further workup confirmed that he has a sensorimotor, primarily demyelinating, polyneuropathy leading to a diagnosis of Guillain Barre’ Syndrome should be a standard in patients who present with polyneuropathy. Early recognition and diagnosis will guide us to the proper management and help improve the patient and their family’s quality of time.

Research - Department of Medicine

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