Distal renal tubular acidosis a case report / Leda O. Villapando.

By:

Villapando, Leda O., MD [author]

Language: English Publication details: Fairview, Quezon City Department of Medicine, FEU-NRMF, 2011Description: (in folder)Content type:

text

Media type:

unmediated

Carrier type:

volume

LOC classification:

MED20110003

Summary: ABSTRACT: Distal renal tubular acidosis is one of the constellations of syndromes involving defects in tubular acid transport. It is characterized by hypokalemia, metablic acidosis, inability to lower urine pH below 5.5 despite an acid load, nephrocalcinosis and nephrolithiasis. Clinically, patients may be asymptomatic or they may present with overt acidosis and hypokalemia. This is the case of XY, a 26 year old male who presented with recurrent lower extremity weakness and hypokalemia. There was no history of vomiting, diarrhea, intake of any medications or carbohydrate loading prior to onset of symptoms. Laboratories done showed metabolic aciidosis, increased urine pH and elevated 24 hour urinary potassium. Further work-up was then done using the ammonium chloride test. Patient tested positive with his serum HC03 level decreasing to less than 5 meq the baseline and his urine pH persisting to be more than 5.3 He was then successfully treated potassium citrate 10 mg/tab one tab three times a day.

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Research	Far Eastern University - Nicanor Reyes Medical Foundation Research	MED20110003 (Browse shelf(Opens below))	Available		R000175

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Includes appendices and bibliographical references.

ABSTRACT: Distal renal tubular acidosis is one of the constellations of syndromes involving defects in tubular acid transport. It is characterized by hypokalemia, metablic acidosis, inability to lower urine pH below 5.5 despite an acid load, nephrocalcinosis and nephrolithiasis. Clinically, patients may be asymptomatic or they may present with overt acidosis and hypokalemia. This is the case of XY, a 26 year old male who presented with recurrent lower extremity weakness and hypokalemia. There was no history of vomiting, diarrhea, intake of any medications or carbohydrate loading prior to onset of symptoms. Laboratories done showed metabolic aciidosis, increased urine pH and elevated 24 hour urinary potassium. Further work-up was then done using the ammonium chloride test. Patient tested positive with his serum HC03 level decreasing to less than 5 meq the baseline and his urine pH persisting to be more than 5.3 He was then successfully treated potassium citrate 10 mg/tab one tab three times a day.

Research - Department of Medicine

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